Please use this identifier to cite or link to this item: https://scholarhub.balamand.edu.lb/handle/uob/7599
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dc.contributor.authorAl Hageh, Cynthiaen_US
dc.contributor.authorO'Sullivan, Siobhánen_US
dc.contributor.authorHenschel, Andreasen_US
dc.contributor.authorAbche, Antoineen_US
dc.contributor.authorHantouche, Mireilleen_US
dc.contributor.authorIakovidou, Nantiaen_US
dc.contributor.authorIssa, Talyen_US
dc.contributor.authorChacar, Stephanieen_US
dc.contributor.authorNader, Monien_US
dc.contributor.authorZalloua, Pierreen_US
dc.date.accessioned2024-10-23T07:36:06Z-
dc.date.available2024-10-23T07:36:06Z-
dc.date.issued2024-10-12-
dc.identifier.urihttps://scholarhub.balamand.edu.lb/handle/uob/7599-
dc.description.abstractSevere coronary artery disease (CAD) represents an advanced arterial narrowing, often associated with critical complications like myocardial infarction and angina. This study aimed to comprehensively investigate determinants of severe and multi-vessel CAD manifestations.en_US
dc.language.isoengen_US
dc.publisherBioMed Central Ltden_US
dc.subjectAPOC1en_US
dc.subjectPHACTR1en_US
dc.subjectMultivessel CADen_US
dc.subjectSevere CADen_US
dc.titlePHACTR1 and APOC1 genetic variants are associated with multi-vessel coronary artery diseaseen_US
dc.typeJournal Articleen_US
dc.identifier.doi10.1186/s12944-024-02327-2-
dc.identifier.pmid39395990-
dc.identifier.scopus2-s2.0-85206123427-
dc.identifier.urlhttps://api.elsevier.com/content/abstract/scopus_id/85206123427-
dc.contributor.affiliationDepartment of Electrical Engineeringen_US
dc.contributor.affiliationFaculty of Medicineen_US
dc.description.volume23en_US
dc.description.issue1en_US
dc.date.catalogued2024-10-22-
dc.description.statusPublisheden_US
dc.identifier.openURLhttps://pubmed.ncbi.nlm.nih.gov/39395990/en_US
dc.relation.ispartoftextLipids in Health and Diseaseen_US
crisitem.author.parentorgFaculty of Engineering-
Appears in Collections:Faculty of Medicine
Department of Electrical Engineering
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